Medical research is fundamental to our health and economic prosperity! We at SmartTots are grateful that Congress has made medical research a national priority through supporting the National Institutes of Health (NIH) and passage of the 21st Century Cures Act. But, without sustained, predictable funding for NIH, tomorrow’s cures may never come. Join SmartTots and urge lawmakers to increase the NIH budget in FY2018 by $2 billion and provide hope to millions of Americans living with life-threatening and chronic diseases. Participate in the #RallyMedRes. www.rallyformedicalresearch.org
- Overexpression of lncRNA Gm15621 alleviates apoptosis and inflammation response resulting from sevoflurane treatment through inhibiting miR-133a/Sox4.
- Role of autophagy in sevoflurane-induced neurotoxicity in neonatal rat hippocampal cells.
- The positive allosteric modulation of GABAA receptors mRNA in immature hippocampal rat neurons by midazolam affects receptor expression and induces apoptosis.
- Euxanthone Ameliorates Sevoflurane-Induced Neurotoxicity in Neonatal Mice.
- Toxicity mechanism of sevoflurane in neural stem cells of rats through DNA methylation.